- June 11, 2013 at 9:55 pm #106113
Sonic BoomMemberTopics: 15Replies: 52
I was just wondering if the probiotic strains (especially L. Acidophilus) from kefir/yogurt remain in our mouths, or if they are somehow “dormant” until digestion time. I was rather shocked as I read the cause of odontosis:
There are three members of the odontosis family; three more-or-less recognizable
progressions of what is in reality the same disease. Since they occur (usually) in order, let’s look
at them in the pattern people usually suffer them:
First signs of odontosis show up in children as cavities. Remember, now, that cavities are
symptoms. Forget anything you may have been told, or assumed, that leads you to think of
cavities as a disease, because they are not. They are symptoms of the initial form of odontosis,
which Oramedics Fellows have named “Cariosis.”
The term cariosis comes from your dentist’s word for cavities, “caries;” “–osis” on the end of
a word simply implies a disease process. (Incidentally, odontosis is a word derived from the
Greek “odontos”—teeth—and the same—osis ending, meaning disease.)
The second stage of odontosis is normally gum irritation and minor infection. Dentists call
the symptoms of this “gingivitis,” from the Latin word for gums, “gingivae.” The “itis” ending
implies inflammation or infection. Oramedics Fellows know this disease as “gingivosis.” Bear
with this, because we’re not simply playing word games, some kind of semantics shuffleboard.
9It’s critical for your understanding of this whole subject, and ultimately your oral health, to
understand that the “old” names and methods were all oriented toward symptoms. The Oramedics
names relate to diseases, and it’s these diseases we want to understand and destroy.
So we’ve introduced the family, “odontosis.” and two of its members: “Cariosis” and
“gingivosis.” The third member of the family is the sneakiest, the deadliest; the one that will rob
you of all your teeth and much of your health. This bad actor’s name is “Periodontosis.” Dentists
actually agree that this is a disease, and they call it periodontal disease, or sometimes
periodontitis. A more popular layman’s term for it is pyorrhea.
We want you to think of the three members of the family as separate, because they occur in
progression and have different symptoms; different effects on your oral and physical health.
But we also want you to develop the understanding that while they are separate in nature, all three are
actually caused by the same thing and all three, in reality, are extensions of the one disease now
known as odontosis.
This disease begins with germs that are usually found in the human mouth sometime after
birth. We don’t have these at first, but they are introduced shortly afterward in a number of ways.
For example: Mama wants to feed her baby some mush or baby oatmeal, but she’s a loving
mother: She wants to make sure it isn’t too hot. What does she do? Naturally, she dips the spoon
into the dish, daintily “samples” the mixture to make sure it’s not too hot (or sweet, or cold, or
bitter…)—and then she gives it to the baby. She also infects the child with the germs of odontosis.
These germs are strains of Streptococcus mutans and Lactobacillus acidophilus. There—you
see? Part of the “mystery” is already gone: The truth about dental disease—odontosis—begins
with the knowledge that we have identified the cause. It isn’t an unknown fear: its well-known
microbes. You’ll be hearing a lot more about Strep. mutans and Lacto. as you continue this book.
Most researchers agree that when these germs are free-floating in the mouth, or disorganized,
they have little effect on oral or physical health. In fact, you may recall reading somewhere that
Lactobacillus germs might even have a beneficial role elsewhere in the body, such as in digestion.
Strep. mutans has no beneficial role (that science is aware of but it is—when disorganized—a
relatively harmless germ because the body is so well protected against its invasions.
The way these germs cause odontosis begins very subtly, almost insidiously. Some of them
manage to come into close association where their waste products form a residue that the natural
“washing” action of saliva doesn’t remove. These waste products include materials called
“dextrans,” which are sticky and virtually transparent. This sticky substance adheres to teeth and
forms a film called “plaque.”
Those who brush their teeth with any frequency will not have this plaque in evidence when
they look into a mirror: The teeth that are visible are pretty and clean. It’s the plaque you don ‘t
see that causes problems. And this would be a good place to remind you that since the U.S.
Health Department says 98 out of 100 Americans are suffering from odontosis, and that two
percent of the public appears to be immune, the statistical implication is that nobody knows how
to brush his teeth well enough to remove both the seen and the unseen plaque.
When plaque adheres to teeth it provides a natural home for colonies of Strep. mutans and
Lacto. Now these germs are organized. The mouth is a perfect germ incubator: The temperature is
a near-constant 98.6 degrees with permanent moisture and, from a germ’s-eye view, a neverending abundance of food.
Comfortably nestled in the plaque film, warm and moist and well-fed, the colonized bugs
achieve explosive growth. As new germs appear by the hundreds of thousands, all of them
10become dextran-generators; they are all plaque-builders. The cycle becomes apparent: More
germs, more tightly organized; larger colonies creating more plaque to harbor even more germs…
The insidious part of all this is that when the germs have developed a colony of metropolitan
size—the New York City of “strep” and “lacto” bugs—we still can’t even see it; don’t really know
Plaque can be seen, particularly when a person has been lax in caring for his teeth over a
period of time. When enough of the substance has built up on visible surfaces of the teeth, it
appears as a whitish-yellow mat; a layer of “gunk” on the teeth. The tongue can feel plaque, in
this amount, as a “furry” sensation.
Seen or unseen, when germs colonize and organize in plaque they achieve an anaerobic
environment. This state, shielded from oxygen and atmosphere, is the environment these bugs
like best of all. Now they begin secreting more than dextrans: They generate acids.
When the germs’ diet contains sugar, acid production increases. When you eat raw sugar, as
in candy, the acid production increases instantly, enormously; and doesn’t taper off for several
hours. Of course, that’s why dentists advise us to avoid sugar and, in these conditions, that is
really good advice.
The point is: What are we doing under these conditions? A disease process is at work; germs
are invading our health. Sugar doesn’t create the condition, it simply makes it worse. Why
eliminate sugar without eliminating the condition that makes sugar “bad for you?”
The acid produced by the germs is trapped between the surface of the teeth (enamel) and the
layer of plaque film. Here the acid begins degrading the calcium-based tooth enamel, which gets
weaker and weaker. This process is known as a carious lesion. In its early development it can’t
properly be called a cavity; but it will become a cavity when the outer enamel is broached and the
softer “insides” of the tooth are exposed to germs, acids and debris.
This whole process is “cariosis”—the disease which produces the symptoms called cavities. It
is during the development of this state that the second stage has its beginning, also.
Gingivosis begins when the plaque film hardens at and slightly below the line where the
gums meet the teeth. This hardening material is called calculus or tartar. It will eventually
resemble the deposits at the bottom of an old teakettle; will get very hard and tenaciously
attached to the teeth, and its surface will be sandpaper-rough,
This forms a natural “dam” which interferes with the natural washing action of saliva. Germs
colonize beneath the gums; more plaque and acid are produced and the calculus continues to
build up and harden in place.
The gum tissue is only one cell thick. It is an incredibly strong tissue for its thickness, and it
has an extremely important job to do, about which more will be said later. No matter how strong
this tissue may be, it is no match for the rough surface of the calculus and the physical action of
chewing, brushing and so on.
Sooner or later the gum tissue gets “holes” in it and opens avenues for infection. The clean,
wholesome pink color is replaced by a more reddish tone and the gums begin to bleed easily.
Congratulations: You now have active cariosis and gingivosis at the same time!
And now you are a prime candidate for stage three: Periodontosis. The disorder beneath the
gums grows steadily worse. Now “pockets” form between the gums and the teeth, reaching
deeper and deeper toward the base of the tooth where it is set into the jawbone.
Tiny, delicate filaments called “connective tissues” anchor the teeth to the gums, very
11reminiscent of guy wires on telephone poles. These tissues are not able to withstand the combined
assault of germs, physical abuse, acids and decaying debris. They are destroyed.
When enough of these tissues are destroyed, the gums sag away from the teeth; the teeth have
no lateral support and become loose in their bone sockets. Often the bone itself will be attacked,
resulting in bone infection. Obviously, all the teeth are ready to fall out, or be pulled out.
It’s denture time, now.
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