Reply To: genital irritation and candida….some questions

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survivor619 wrote: I have been having problems with itchiness, excessive sweatiness, and redness in the groin and genital area. that is how all of my candida symptoms started (before the “ibs”, prostatitis, fatigue etc). inspite of antifungal creams, the problem won’t clear up, is this because the candida is still living in my digestive tract? does the candida in the rest of my body have to go away in order for the candida on my genitals to clear up? sorry if that sounds stupid but i have to get an answer since it’s been really bothering me. a dermatologist said that I have overactive sweat glands in my groin area and that botox may help but it is expensive. I have not yet pursued that because I wonder if i actually do have overactive sweat glands in my groin, or if all of the problems in that area is related to candida? that is where candida usually starts anyways right?

You have to recover your immunity against candida to eradicate it.
Your immune system is tolerating the yeast.

Read this carefully :

People can become
unresponsive to an antigen by exposing them more or
less continually to this antigen. This may be carried
out with very high doses of antigen—called “highzone
tolerance”—or with very low doses, “lowzone
tolerance.” High-zone tolerance tends to
render unresponsive both T- and B-cells, whereas
low-zone tolerance weakens or blocks the T-cell
response only. Thus the cellular immune response
(T-cell) is more easily blocked, and such blockage
is easier to sustain than is the humoral (B-cell)
response. Furthermore, unresponsiveness (or
“tolerance”) is easier to induce with soluble than
with aggregated antigens.
Thus immunologic tolerance is most readily
achieved by more or less continual exposure to a
soluble antigen, with T-cell paralysis being much
easier to achieve and sustain.
When mucous membranes are chronically infected
by yeast, cells of the immune system are exposed
continually to Candida antigens, thus satisfying the
conditions for tolerance induction. This weakened
immunologic response allows the yeast to thrive in
the tissues, and a vicious cycle is established. An
analagous situation has been reported in
lepromatous leprosy. In this, the widely
disseminated form of leprosy, impairment of the
immune response has been demonstrated in vivo by
reversion to negative of the lepromin skin test, and
in vitro both by impairment of the lymphocyte
transformation test on exposure to the leprosy
bacillus or phytohemagglu-tinin, and release of the
migration inhibitory factor on exposure to the
bacillus. In the tuberculoid form of leprosy,
however, this does not occur. This is the localized
form of the disease, which carries a good
prognosis. Thus widespread involvement is
associated with impairment of the immune
With Candida infection, as with leprosy, there
may be for each individual a critical point beyond
which the total area of involvement is such that the
antigenic stimulus paralyzes rather than stimulates
the immune system, particularly the T-cell compartment.
Until this state of immunologic
unresponsiveness is broken, drug therapy of the
yeast infection will be less than fully effective.