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I am against drinking substances that alkalize the intestines.
There is only one thing I could take temporary, Cimetidine. (Tagamet) It may change the things regarding to a persistent fungal infection.
The bacterial flora antagonism is caused by the production of Acetic and Lactic Acids by some type of bacterias. It looks like it inhibits the growth of Candida and avoids its morphogenesis to a pathogenic state.
Candida has influence in the re-colonization of the gut after antibiotic intake. It avoid Lactic Acid producer re-colonization by inhibitory mechanisms.
This is the result of Lactic Acid inhibitory activity at different Phs. This suggest that at alkaline PH there isn’t protection from Lactic Acid. So, consider carefully rising the intestinal Ph to an alkaline state. The article stays there isn’t inhibition by Lactic Acid at Ph above 4.5. Well, this open more questions because the normal Colon Ph is between 5.5 and 7. I mean the large bowel where candida growths more than any other place. This may suggest there are other inhibitory mechanisms in the intestines no linked to the Lactic Acid production.
Effects of pH and Temperature on Anti-C. albicans Activity
The anti-C. albicans activity was found to be heat stable. Concentrated
supernatant autoclaved for 15 min at 121°C retained full inhibitory activity against C.
albicans growth. The activity was stable at pH values that were between 2.5-4.0 but
rapidly decreased between pH 4.0-4.5. No inhibitory activity was detected at a pH
above 4.5. The activity was regained after readjustment of the pH to the starting value
(pH 3.5). Initially, a cross streak method was used to evaluate the anti-C. albicans
activities. The pH value in the zone was approximately 3.5 to 3.8, suggesting a limited
contribution of undissociated lactic acid to the inhibitory effect. However, the
reduction in anti-C. albicans activity of the culture filtrates at pH values exceeding 4.0
indicates synergistic effects between lactic acid and other antifungal substances. The
possibility of increased desorption of antifungal substances from the bacterial cells at
very low pH values suggests further very complex interactions between the antifungal
effects of LAB and the pH (20).