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jameskep;56116 wrote: If S. cerevisiae can morph like candida than why couldn’t S.boulardi?
Do you have research that shows S.Boulardi can’t morph?
I haven’t read any document about S.Boulardii morphogenesis. Anyway, candida albicans is a pathogen, S. Cervisiae and S. Boulardii aren’t. The fact they can morph doesn’t mean they become pathogens, at least until there be proofs of it.
The fact that candida albicans is polymorphic doesn’t mean the pathogen is only the elongated form. Candida Albicans is a pathogen in ANY form. Morphogenesis increase its virulence, tissue invasion, and growth factor. However, yeast cells are also infectious organisms and are massively present during an infection. Morphogenesis is regulated by some mechanisms still not too clear. There are many things involved. Nutrient deprivation and serum favor morphogenesis. Ph, temperature, and the environment also influence its phenotype changes.
Sporadic candida albicans cells living in the intestines aren’t enough to cause considerable damage and symptoms to the human body. This is the case of a “normal” balanced flora. These non-forming colonies candida albicans single cells are controlled by antifungal substances produced by the anaerobic bacterial flora and the immune system. After some amount of antibiotics, this mechanism is disrupted and candida growths. It growths as individual cells and possibly forming germ tubes and mycelia. The result is an infection where different forms are present. This happens because candida albicans is an opportunistic pathogen, a pathogen controlled by the friendly bacterial flora and the immunity. This isn’t the case of S. Boulardii so far. It is considered a beneficial yeast until the opposite be demonstrate.
You can read in the web about candida switching from yeast to fungus as the cause of this syndrome because there is ignorance about the reality. There is lack of knowledge written everywhere about this syndrome. It has been hard for me to learn and to correct many myths I have read about it. Still, I don’t know all, but I have had the opportunity of sharing documents and knowledge with two recognized Mycologists who have explained me many things.
Yes, it is TRUE candida albicans gain virulence, mobility and tissue invasion after morphing from a single yeast to an elongated form. However, candida albicans is “commensal” only when there are sporadic cells living in the human colon and other mucosal areas. When the conditions are ready, these sporadic cells can GROWTH and become infectious not matter what phenotype change they adopt. Of course, more virulence means a more severe infection.